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Venous content CvO ; and saturation SvO ; were measured using blood analyzers Instrumentation Laboratories ; . Total pulmonary re sistance TPR ; was calculated by the formula TPR dynes.s.cm' ; pulmonary artery pressure PAP ; x 80 cardiac output. Pulmonary vascular resistance PVR ; was calculated by the formula PVR PAP " WP ; x cardiac output where PWP pulmonary wedge P pressure mm Hg
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Financial support for the study was provided by Solvay Pharmaceuticals GmbH and Aventis Pharma Germany. Solvay Pharmaceuticals GmbH provided study medication. All data were collected and analyzed independent from the sponsors, and the study was designed, conducted, analyzed, and interpreted by the investigators independently of all sponsors. The presentation of data are the intellectual property of the MOSES writing committee, and the manuscript was reviewed. All members of the MOSES writing committee contributed to the writing of this article.
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Complete the following physician reimbursement table. To the extent you are subcontracting or leasing provider networks and reimbursement methodologies vary by network, then please answer this question separately for each subcontracting arrangement. Name of Network.
Relative to sodium. The increase in plasma osmolality, induced by a rising plasma sodium concentration, results in water movement out of the cells into the extracellular fluid, and the resultant cellular dehydration of brain cells is responsible for the dominant clinical symptoms lethargy, seizures, coma ; . The severity of hypernatraemic symptoms is related primarily to the rate of rise in plasma osmolality rather than to the absolute degree of hypernatraemia [2]. Impairment of AVP production, release or action can lead to profound water deficits and to polyuria and phenobarbital.
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