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Fig 3 Propofol target plasma concentration TCI ; , bispectral index, blood concentration of propofol, heart rate HR ; and mean arterial pressure MAP ; before pre ; and after hold ; clonidine administration and after achieving the pre-BIS-value post ; with possible reduction of propofol in patients treated with i.v. clonidine or placebo meanTSD ; . Signicant difference from `pre' condition within-group comparison, after Bonferroni correction, P 0.017 ; . Signicant difference from `hold' condition within-group comparison, after Bonferroni correction, P 0.017 ; . * Signicant difference between groups after Bonferroni correction, P 0.017. Ribchester, R.R., Thomson, D., Wood, N.I., Hinks, T., Gillingwater, T.H., Wishart, T.M., Court, F.A. and Morton, A.J. 2004 ; Progressive abnormalities in skeletal muscle and neuromuscular junctions of transgenic mice expressing the Huntington's disease mutation. Eur J Neurosci, 20, 3092-3114.

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To the hCG group data not shown ; , indicative of an early malfunction of the trophoblast irrespective of the luteal phase support given. In earlier studies, intramuscular progesterone injections Fauser et al., 2002 ; , micronized progesterone vaginally Bracero et al., 2001 ; , intramuscular progesterone injections and estradiol orally Itskovitz et al., 2000 ; was used for luteal support. Penarrubia et al. 1998 ; showed that luteal hCG support can overcome luteal phase insufficiency after triggering of ovulation with GnRH agonist in gonadotrophin-stimulated cycles, a concept which is applicable in patients at low risk of OHSS. An alternative to this model could be a single bolus of hCG, 1500 IU s.c., following the oocyte retrieval. In IUI cycles in which ovulation was triggered with 0.1 mg triptorelin, Emperaire et al. 2004 ; showed that the luteal phase was completely restored after a single injection of 1500 IU hCG given 12 h after triggering of ovulation. A study is currently being undertaken in our clinic focusing on optimizing the luteal phase support. In summary, the present data suggest for the first time a significant beneficial effect of the mid-cycle FSH surge on oocyte maturation. The use of a GnRH agonist for triggering of ovulation in FSH GnRH antagonist IVF ICSI cycles, however, disclosed a detrimental effect on clinical outcome parameters, presumably due to a luteal phase inadequacy, despite luteal support. Future studies are needed to explore the type of luteal phase support needed before this model is clinically acceptable.

Siracusa A, Desrosiers M, Marabini A. Epidemiology of occupational rhinitis: prevalence, aetiology and determinants. Clin Exp Allergy 2000; 30: 15191534. Malo JL, Lemiere C, Desjardins A, Cartier A. Prevalence and intensity of rhinoconjunctivitis in subjects with occupational asthma. Eur Respir J 1997; 10: 15131515. Moscato G, Galdi E, Perfetti L. Early detection of occupational asthma and reactive airway disease. Monaldi Arch Chest Dis 2000; 55: 6669.

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Table 4. Summary of clinical response. Patient group n CR % ; PR % ; and PR % ; 21 57 ; 95% CI CR and PR ; 41%-73% 44%-76.

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Fig. 1. Key pharmacokinetic parameters for patients with rejection, CsA nephrotoxicity and liver toxicity during the first month posttransplant. * P 0.05, * P 0.01.

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