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Frequently ulcerate, and lymphomas involving the gastrointestinal tract may be associated with massive hemorrhage.
P until July 2002, based on the accumulated evidence, estrogen therapy was gaining in importance for treating the most common symptoms of estrogen decline in menopausal women: vasomotor symptoms, urogenital atrophy, and bone loss. The use of oral hormone therapy in postmenopausal women has been profoundly reduced as a result of the Women's Health Initiative WHI ; studies of the effects of oral estrogen therapy conjugated equine estrogen 0.625 mg d ; and oral estrogen progestin therapy conjugated equine estrogen 0.625 mg d and medroxyprogesterone acetate 2.5 mg d ; . As an antidote to the potential risks of oral therapy, the transdermal route of administration is gaining significant attention, and may be of benefit to many patients taking hormone therapy.
The Rotterdam Symptom Checklist. Br J Cancer 1990; 62: 10348. Ravaioli A, Buda P, Fava C, Paci E, Tononi A, Riva N, et al. Assessment of the RSCL quality of life instrument during chemotherapy in an Italian setting. Qual Life Res 1996; 5: 4915. Diggle PJ, Linang KY, Zeger SL. Analysis of longitudinal data. Oxford U.K. ; : Clarendon Press; 1994. Montazeri A, Gillis CR, McEwen J. Quality of life in patients with lung cancer: a review of literature from 1970 to 1995. Chest 1998; 113: 46781. Heymsfield SB, McManus CB. Tissue components of weight loss in cancer patients. A new method of study and preliminary observations. Cancer 1985; 55: 23849. Anonymous. Randomised trial of four-drug vs less intensive two-drug chemotherapy in the palliative treatment of patients with small-cell lung cancer SCLC ; and poor prognosis. Medical Research Council Lung Cancer Working Party. Br J Cancer 1996; 73: 40613. Beal JE, Olson R, Lefkowitz L, Laubenstein L, Bellman P, Yangco B, et al. Long-term efficacy and safety of dronabinol for acquired immunodeficiency syndrome-associated anorexia. J Pain Symptom Manage 1997; 14: 714. Popiela T, Lucchi R, Giongo F. Methylprednisolone as palliative therapy for female terminal cancer patients. The Methylprednisolone Female Preterminal Cancer Study Group. Eur J Cancer Clin Oncol 1989; 25: 18239. Schneeberger AL, Thompson RT, Driedger AA, Finley RJ, Inculet RI. Effect of cancer on the in vivo energy state of rat liver and skeletal muscle. Cancer Res 1989; 49: 11604. Okajima F, Tokumitsu Y, Kondo Y, Ui M. P2-purinergic receptors are coupled to two signal transduction systems leading to inhibition of cAMP generation and to production of inositol triphosphate in rat hepatocytes. J Biol Chem 1987; 262: 1348390. Asensi M, Lopez-Rodas A, Sastre J, Vina J, Estrela JM. Inhibition of gluconeogenesis by extracellular ATP in isolated rat hepatocytes. J Physiol 1991; 261: R15226. Keppens S, De Wulf H. Characterization of the liver P2-purinoceptor involved in the activation of glycogen phosphorylase. Biochem J 1986; 240: 36771. Wang P, Ba ZF, Morrison MH, Ayala A, Dean RE, Chaudry IH. Mechanism of the beneficial effects of ATP-MgCl 2 following traumahemorrhage and resuscitation: downregulation of inflammatory cytokine TNF, IL-6 ; release. J Surg Res 1992; 52: 36471. Moldawer LL, Rogy MA, Lowry SF. The role of cytokines in cancer cachexia. JPEN J Parenter Enteral Nutr 1992; 16: 43S49S. Barber MD, Fearon KC, Ross JA. Relationship of serum levels of interleukin-6, soluble interleukin-6 receptor and tumour necrosis factor receptors to the acute-phase protein response in advanced pancreatic cancer. Clin Sci Colch ; 1999; 96: 837.
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4 MMI Training of Tied-Mixture Density Hidden Markov Models 4.1 Introduction . 4.2 Recognition System Overview . 4.2.1 Database and Feature Extraction . 4.2.2 Recogniser Structure . 4.3 Parameter Tying in HMMs . 4.4 MMI Optimisation Algorithms for TDHMMs . 4.4.1 Extended Baum-Welch Algorithm . 4.4.2 Gradient Descent Algorithm . 4.4.3 QuickProp Algorithm . 4.4.4 Comparison of MMI Optimisation Algorithms . 4.5 MMI Training Performance and Parameter Discriminability . 4.6 Effects of Parameter Tying in MMI Training . 4.7 MMI Training in Detailed-Match 4.8 Discussion and Summary . Thai Character Recognition 5.1 Introduction . 5.2 Characteristics of the Thai Language . 5.3 Previous Research in Thai Character Recognition 5.3.1 Pre-Processing Techniques . 5.3.2 Printed Thai Character Recognition . 5.3.3 Handwritten Thai Character Recognition . 5.3.4 On-line Thai Handwriting Recognition 10.
Public agency offering STD and Confidential and Anonymous HIV testing, prevention, immunization, and TB skin testing. Appointments are preferred but will take walk-ins. Agencia publica ofrece examenes confidenciales y annimos de VIH y STD enfermedades de transmisin sexual ; , prevencin, vacunas y examenes de piel de TB. Referencias con cita previa!
FIG. 6. Immunohistochemical staining of HSP90 in myometrial smooth muscle cells is stronger in the proliferative phase A ; than in the secretory phase B ; of the menstrual cycle. Magnification, 200 and dss.
Road around the lake. As a Christmas gift, the Carlsons brought us a set of bells for Rowdy's harness, and so he jingle-jangled his bells as he trotted along through snowfilled woods of balsam fir and white birch, and over a particularly picturesque section that I had built the previous summer especially for such occasions and which I had christened "Rowdy's Road." Rowdy was his usual self -- tail in the air, head high, nostrils dilated, glancing about as though on the alert for some unknown danger, skittishly prancing a little sideways on occasion at some twig or branch along the way as though it was about to leap at him with bared fangs. Other people might say that he "shies, " but I always interpreted his general behavior as the result of his being the equine equivalent of a smart aleck. Anyway, he blended a spirited performance into the quiet beauty of the winter woods, eliciting a nostalgia for a past way of life -- a nostalgia that deepens and expands into a mystic communion with the whole of the living universe, past and present. After I had put Rowdy back in his stall, we men decided to luxuriate with a sauna. Janet was particularly amused when three of us males came trotting from the bedroom through the living room with only a towel about our loins on our way outside, barefoot through the snow, to the sauna. Whether it seemed incongruous with the Christmas season to her, or that she was titillated by this brash violation of her Puritan upbringing, I haven't been able to decide. Festivities were capped that evening by lighting the candles on the Christmas tree. Without electricity, we were spared electric tree lights. The trouble with electric lights is that they are on hours on end, day.
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Tics that can promote important differences in the final response Bazzaz, 1990, El-Gengaihi 1998, Briskin 2000 ; . Thus, it seems clear that the increase of CO2 increases primary productivity, but it is more difficult to establish the response of secondary metabolism, above all when the interest is to obtain specific compounds with high pharmacological value Biel et al. 2003 and dulcolax.
BPE has both static increased tissue mass ; and dynamic increased smooth muscle tone ; components in the prostate.10 Medical management using 5-alpha-reductase inhibitors 5-ARIs ; inhibits prostatic growth, whereas alpha-1-adrenergic receptor antagonists alphablockers ; reduce the tone of prostatic smooth muscle. Post-void residual PVR ; volume may be used as a potential predictor of Treatment Response in Male Lower Urinary Tract Symptoms Varies Practitioners should consider the possible involvement of bladder dysfunction in male LUTS, as interventions that target the prostate often leave a subset of patients with inadequate symptom control. It is shown that urgency, frequency and nocturia persist in 19% of men after TURP.11 Interestingly, this subset of storage LUTS comprises the same symptoms as OAB. Also, Thomas et al.12 have recently shown that even in men who have had resolution of OAB symptoms after TURP, 48 of 58 83% ; experienced return of their storage symptoms at long-term follow-up mean 12.6 years after surgery ; . Lee et al.8 found that treatment response may vary in men with LUTS due to different conditions. After three months of treatment with doxazosin alpha-blocker ; , 60 79% ; with BOO and 24 35% ; with BOO + DO reported symptomatic improvement. In those patients with no improvement, 32 of 44 73% ; with BOO + DO improved after combined therapy with tolterodine antimuscarinic ; . Antimuscarinics and Acute Urinary Retention Undertreatment of Overactive Bladder in Male Lower Urinary Tract Symptoms Despite equal prevalence of OAB, fewer men than women are treated with antimuscarinics. This can be explained by the alternatives in potential pathophysiology: male LUTS can be caused solely by bladder dysfunction or mixed with prostatic pathology. This results in a complex clinical presentation of OAB symptoms in combination with other LUTS voiding symptoms ; . Also, significant regional and country differences in.
Where a problem arises--have been in part demolished. Particularly within regions--notably Europe and North America--agencies do communicate with one another in sufficient detail to provide help and advice. A basic problem that remains and indeed has become more acute with the expansion of the world community is that of assisting developing countries. Much of the drug regulatory information as it currently exists is attuned primarily to the needs of industrialised countries where the protection of intellectual property and industrial interests appears sometimes to weigh as heavily as the interests of public health--sometimes more heavily than these. In many developing countries--certainly those at the lowest level of development--it is true that the bulk of the population are treated with a basic list of `Essential Drugs' according to WHO principles, and that these are well-established products, the efficacy and safety of which are well recognised. Alongside these `essential' products, however, almost every country has a certain trade through the private sector in drugs of much more recent origin, some only newly registered even in Europe and the United States. In countries at a middle level of development--including many in Africa and Asia as well as those emerging from the former Soviet sphere of influence-- the turnover in such `new' drugs may be considerable, yet regulatory law, procedure and experience is often poorly developed. It is here that the unhappy influence of the `confidentiality clauses' in the West is most clearly felt. In Russia, in the Middle East, in Latin America and in South-East Asia one still too often encounters drugs on sale which in the industrialised West have for good reasons been banned, restricted, or refused entry to the market. It is there that the enlightened policy-maker should be looking. Confidentiality clauses, intelligently formulated and applied--have their place in drug regulation; but the time has come to consider anew their proper scope, and to recognise those situations where--viewing the world's health as a whole--they are doing more harm than good. One way ahead is offered by the formulation of confidentiality clauses in the new legislation of some developing countries, which are less rigid than those traditionally employed. Uganda's 1993 Drug Policy and Authority Statute, for example, binds the National Drug Authority to secrecy `. except in so far as the provisions of the Act require or the Authority has determined that information on particular matters shall be published or made available to other specified parties'. That at least opens the door to release of data in the public interest. Perhaps it is actually the case that, having suffered long enough the consequences of the rigid confidentiality clauses devised by the industrialised west, the developing world is now equipped to point the way to more reasoned policies in the future and duragesic.
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1184. 61. Crespo M, Bosch F, Villamor N, et al. ZAP-70 expression as a surrogate for immunoglobulin-variable-region mutations in chronic lymphocytic leukemia. N Engl J Med. 2003; 348: 17641775 Wiestner A, Rosenwald A, Barry TS, et al. ZAP-70 expression identifies a chronic lymphocytic leukemia subtype with unmutated immunoglobulin genes, inferior clinical outcome, and distinct gene expression profile. Blood. 2003; 101: 49444951 Ibrahim S, Keating M, Do KA, et al. CD38 expression as an important prognostic factor in B-cell chronic lymphocytic leukemia. Blood. 2001; 98: 181-186. Chang CC, Cleveland RP. Conversion of CD38 and or myeloid-associated marker expression status during the course of B-CLL: association with a change to an aggressive clinical course. Blood. 2002; 100: 1106. Messmer D, Messmer B, Albesiano E, et al. Direct measurement of B-CLL cell production and turnover in vivo. Blood. 2002; 100: 169a Hellerstein M, Neese R. Mass isotopomer distribution analysis at eight years: theoretical, analytic, and experimental considerations. J Physiol. 1999; 276: E1146-E1170 67. Zupo S, Cutrona G, Mangiola M, Ferrarini M. Role of surface IgM and IgD on survival of the cells from B-cell chronic lymphocytic leukemia. Blood. 2002; 99: 2277-2278. Broker BM, Klajman A, Youinou P, et al. Chronic lymphocytic leukemic cells secrete multispecific autoantibodies. J Autoimmun. 1988; 1: 469-481. Sthoeger ZM, Wakai M, Tse DB, et al. Production of autoantibodies by CD5-expressing B lymphocytes from patients with chronic lymphocytic leukemia. J Exp Med. 1989; 169: 255-268 Borche L, Lim A, Binet JL, Dighiero G. Evidence that chronic lymphocytic leukemia B lymphocytes are frequently committed to production of natural autoantibodies. Blood. 1990; 76: 562-569 Silverman GJ, Goldfien RD, Chen P, et al. Idiotypic and subgroup analysis of human monoclonal rheumatoid factors. Implications for structural and genetic basis of autoantibodies in humans. J Clin Invest. 1988; 82: 469-475 Pascual V, Victor K, Spellerberg M, Hamblin TJ, Stevenson FK, Capra JD. VH restriction among human cold agglutinins. The VH4-21 gene segment is required to encode anti-I and anti-i specificities. J Immunol. 1992; 149: 2337-2344. Stevenson FK, Longhurst C, Chapman CJ, et al. Utilization of the VH4-21 gene segment by anti-DNA antibodies from patients with systemic lupus erythematosus. J Autoimmun. 1993; 6: 809-825. Adderson EE, Shackelford PG, Quinn A, et al. Restricted immunoglobulin VH usage and VDJ combinations in the human response to Haemophilus influenzae type b capsular polysaccharide. Nucleotide sequences of monospecific antiHaemophilus antibodies and polyspecific antibodies crossreacting with self antigens. J Clin Invest. 1993; 91: 2734-2743. Zhou J, Lottenbach KR, Barenkamp SJ, Lucas AH, Reason DC. Recurrent variable region gene usage and somatic mutation in the human antibody response to the capsular polysaccharide of Streptococcus pneumoniae type 23F. Infect Immun. 2002; 70: 4083-4091 Diaw L, Siwarski D, Coleman A, et al. Restricted immunoglobulin variable region Ig V ; gene expression accompanies secondary rearrangements of light chain Ig V genes in mouse plasmacytomas. J Exp Med. 1999; 190: 1405-1416 Diamond B, Scharff MD. Somatic mutation of the T15 heavy.
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21. Hylek EM, Singer DE. Risk factors for intracranial hemorrhage in outpatients taking warfarin. Ann Intern Med. 1994; 120: 897-902. Hirsh J, Fuster V. Guide to anticoagulant therapy. Part 2: Oral anticoagulants. American Heart Association. Circulation. 1994; 89: 1469-80. Hirsh J, Dalen JE, Deykin D, Poller L, Bussey H. Oral anticoagulants. Mechanism of action, clinical effectiveness, and optimal therapeutic range. Chest. 1995; 108: 231S246S. Horton JD, Bushwick BM. Warfarin therapy: evolving strategies in anticoagulation. Fam Physician. 1999; 59: 635-46. Lanoxin digoxin ; : Product Prescribing Information. GlaxoWellcome, Inc. Nov, 2000. Accessed online Nov 2, 2001 ; : : us.gsk products assets us lanoxin tablets ' and echinacea.
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22 further evidence citing the potential dangers of marinol exists in the 1995 marinol product insert itself, which warns against giving dronabinol to children and to the elderly although unimed is currently in phase iii testing for approval of marinol in the treatment of alzheimer's patients ; because of the drug's psychoactive effects and efalizumab.
Figure 2. New guidelines to help patients manage their migraine!
The total area on which the future of these monkeys and indeed that of the entire South Eastern Brazilian flora and fauna depends, certainly does not exceed 500, 000 ha. An understanding of the ecology of such animals is vital to any hopes of their surviving in the wild even to the turn of the century. World Wildlife Fund Year Book 1980 - 81 and Red Data Book 1988 and eletriptan.
Ated expression of bcl-2 in B-chronic lymphocytic leukemia cells. Blood 79: 2981, 1992 Mariano MT, Moretti L, Donelli A, Grantini M, Montagnani G, Di Prisco AU, Torelli G, Torelli U, Narni F: bcl-2 gene expression in hematopoietic cell differentiation. Blood 80: 768, 1992 Hanada M, Delia D, Aiello A, Stadtmauer E, Reed JC: bcl2 gene hypomethylation and high-level expression in B-cell chronic lymphocytic leukemia. Blood 82: 1820, 1993 Cory S: Regulation of lymphocyte survival by the bcl-2 gene family. Annu Rev Immunol 13: 513, 1995 Yang E, Korsmeyer SJ: Molecular thanatopsis: A discourse on the BCL2 family and cell death. Blood 88: 386, 1996 Hockenbery DM, Oltvai ZN, Yin XM, Milliman CL, Korsmeyer SJ: Bcl-2 functions in an antioxidant pathway to prevent apoptosis. Cell 75: 241, 1993 Kane DJ, Sarafian TA, Anton R, Hahn H, Butler Gralla E, Selverstone Valentine J, Ord T, Bredesen DE: Bcl-2 inhibition of neural death: Decreased generation of reactive oxygen species. Science 262: 1274, 1993 Lam M, Dubyak G, Chen L, Nunez G, Miesfeld RL, Distelhorst CW: Evidence that BCL-2 represses apoptosis by regulating endoplasmic reticulum-associated Ca2 fluxes. Proc Natl Acad Sci USA 91: 6569, 1994 Martin SJ, Green DR: Protease activation during apoptosis: Death by a thousand cuts? Cell 82: 349, 1995 Chinnaiyan AM, Dixit VM: The cell-death machine. Curr Biol 6: 555, 1996 Henkart PA: ICE family of proteases: Mediators of all apoptotic cell death? Immunity 4: 195, 1996 De Murcia G, Menissier de Murcia J: Poly ADP-ribose ; poly merase: A molecular nick-sensor. Trends Biochem Sci 19: 172, 1994 Kaufmann SH, Desnoyers S, Ottaviano Y, Davidson NE, Poirier GG: Specific proteolytic cleavage of poly ADP-ribose ; polymerase: An early marker of chemotherapy-induced apoptosis. Cancer Res 53: 3976, 1993 Lazebnik YA, Kaufmann SH, Desnoyers S, Poirier GG, Earnshaw WC: Cleavage of poly ADP-ribose ; polymerase by a proteinase with properties like ICE. Nature 371: 346, 1994 Tewari M, Quan LT, O'Rourke K, Desnoyers S, Zeng Z, Beidler DR, Poirier GG, Salvesen GS, Dixit VM: Yama CPP32b, a mammalian homolog of CED-3, is a CrmA-inhibitable protease that cleaves the death substrate poly ADP-ribose ; polymerase. Cell 81: 801, 1995 Nicholson DW, Ali A, Thornberry NA, Vaillancourt JP, Ding CK, Gallant M, Gareau Y, Griffin PR, Labelle M, Lazebnik YA, Munday NA, Raju SM, Smulson ME, Yamin TT, Yu VL, Miller DK: Identification and inhibition of the ICE CED-3 protease necessary for mammalian apoptosis. Nature 376: 37, 1995 An S, Knox KA: Ligation of CD40 rescues Ramos-Burkitt lymphoma B cells from calcium ionophore- and antigen receptortriggered apoptosis by inhibiting activation of the cysteine protease CPP32 Yama and cleavage of its substrate PARP. FEBS Lett 386: 115, 1996 Binet JL, Leporria M, Dighiero G, Charron P, D'Athis P, Vaugier G, Merle-Beral H, Natali JC, Raphael M, Nizet B, Follezou JY: A clinical staging system for chronic lymphocytic leukemia. Cancer 40: 855, 1977 Carballo E, Colomer D, Vives Corrons JL, Blackshear PJ, Gil J: Characterization and purification of a protein kinase C substrate in human B cells. J Immunol 156: 1709, 1996 Mosmann T: Rapid colorimetric assay for cellular growth and survival: Application to proliferation and cytotoxicity assays. J Immunol Methods 65: 55, 1983 Forbes IJ, Zalewski PD, Giannakis C, Cowled PA: Induction and dronabinol.
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Not FDA approved for treatment of ED. Approved and used in Europe. No proof of efficacy safety. 1. Lue TF. N Engl J Med. 2000; 342: 1802-1813. Lue TF et al. In: Lue TF et al, eds. Sexual Medicine: Sexual Dysfunctions in Men and Women. Paris, France: Health Publications; 2004: 605-627. 3. Padma-Nathan H, Giuliano F. Urol Clin North Am. 2001; 28: 321-334. Dula E et al. Eur Urol. 2001; 39: 558-564. Moyad MA et al. Semin Urol Oncol. 1999; 17: 103-110 and elidel.
Chemotherapy CONTRAINDICATIONS hypersensitivity to Dronabinol or any compound or marijuana; patients with history of schitzophrenia, heart disease, hepatic disease, seizure disorders, severe hepatic impairment DOSING Oral: Initial dose- 5mg m2 po 1-3 hours before chemotherapy, then q2-4 hours after chemotherapy, for a total of 4-6 doses Dose escalation- If initial dose ineffective and drug tolerated, increase by 2.5mg m2 increments to a maximum of 15mg m2 dose Caution is needed during dose escalation, since disturbing psychiatric symptoms are more common at higher doses.
Medication Issues Recent focus on drug-drug interactions, drug interactions with food, food components or dietary supplements and the effects of phytochemicals on P450 enzyme activity has caused both clinicians and HIV-positive people to restructure medication scheduling strategies. In addition to medications to combat and manage HIV infection, many patients also take medications for symptom management or nutritional rehabilitation. Symptom management strategies see also MNT section p 22 ; are typically aimed at supporting tolerance and effectiveness of drug therapies as well as quality of life. Nutrition rehabilitation includes nutrient support, exercise, antiretroviral therapy which requires strict adherence to drug regimens 112 ; , prevention and treatment of other infection and or neoplasm, and immune function restoration. The effectiveness and tolerability of some drugs can be affected by the body's nutritional stores. The overall bioavailability and efficacy of a drug can be influenced by an individual's plasma protein concentration as in the case of a highly protein-bound medication ; and by the drug's volume of distribution. Both factors are affected by nutritional status. Information about the actual incidence and severity of food medication interactions is incomplete at this time, especially with many of the newly approved drugs. Consideration of these interactions, as well as drug-drug see p139 ; and drug-herbal see p 220 ; interactions, is an important part of a multi-faceted health care plan. Medications used to try to restore positive nutritional status include vitamins, minerals, appetite stimulants such as megestrol acetate Megace ; or dronabinol Marinol ; , anticytokine therapies such as thalidomide Thalomid ; and anabolic therapies such as growth hormone or anabolic steroids ; . Choosing to prescribe, recommend or use these drugs should be part of a care plan for prevention and treatment of nutritional compromise. Combinations of therapies are likely to be more effective in some cases, such as the use of megestrol acetate with anabolic agents a nd resistance exercise.113 Value of MNT Medical Nutrition Therapy ; MNT to maintain or improve nutritional status is a necessary part of the management of HIV disease. Individualization of fluid, calorie, protein and other nutrient needs with respect to and consideration of cultural aspects, current food habits, nutrition-related beliefs and medical status may determine the patient's ability to manage his her disease. Successful treatment of wasting syndrome, altered and eligard.
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Q. I enjoy outdoor sporting activities during the summer. How can I tell if I suffering a heat related illness? What should i do to prevent it? A. Heat related illness, like sunstroke or heat cramps, happens when the body cannot cool itself. When air temperature goes up, your body stays cool when your sweat evaporates. High humidity slows down this process. If you are not sweating enough your body temperature may rise. This can cause a heat related illness. Symptoms may include headache, dizziness, muscle weakness, cramps or nausea and vomiting. If you are having any of these symptoms, try to get to a shady, cooler area. Slowly sipping water and applying cool, wet cloths should help. Here are some tips to help prevent heat related illness: Wear lightweight, light colored, and loose fitting clothing. Protect yourself from the sun by wearing a hat or using an umbrella. Don't forget the sunscreen and sunglasses. Drink a lot of water before going outside. Drink extra water all day. Avoid tea, coffee, cola or any drink with caffeine ; and all alcoholic beverages. Plan outdoor activities at cooler times of the day-before 10AM or after 6PM. If you have a medical condition, ask you PCP about how to deal with the heat. If you experience any of the following symptoms, you may have Heat Stoke. This is a more serious condition and needs medical attention right away: hot, dry skin but not sweaty, confusion or loss of consciousness, frequent vomiting, shortness of breath or trouble breathing and dss.
1 2 McFadden ER, Jr, Warren EL. Observations on asthma mortality. Ann Intern Med 1997; 127: 142-7. Cowie RL, Revitt SG, Underwood MF, Field SK. The effect of a peak flowbased action plan in the prevention of exacerbations of asthma. Chest 1997; 112: 1534-8 and elmiron.
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